The difference is that the clonus is caused by passive muscle stretching, while the tremor is typically caused by a direct injury to the upper motor neurons in the spinal cord, such as a nerve cell. Spasticity in cloning is the result of an over motor neuron lesion that inhibits the tendon stretching reflex and is distinguished by the absence of muscle flexure, muscle contraction or muscle contractions and muscle cramps.
Although clonus can be caused by many other neurological disorders, treatment is needed for the specific disease that causes it to receive relief. Antifever drugs can also help with fever, as the fever is due to increased muscle activity and not to the underlying disease. It is also thought that paralysis can help by preventing the worsening hyperthermia and rhabdomyolysis that occur in people with spinal cord injuries such as spasticity during cloning and spinal muscular dystrophy.
Many medical professionals use the term “hypertension” alternately with “spasticity,” and some describe it as a convulsion that can occur constantly or suddenly. However, it is hypertonia that is dependent on speed and is amplified by movement, while spasticity is present at rest. Spasticity is described as an increase in muscle activity during movement and cramps, which are constant and sudden and can occur either at rest or during movement.
By scraping the soles, the toes pull, and by the hammer mechanism at the lateral thighs, they are moved in a completely normal downward movement.
The ankle is repeatedly dorsiflexion, which allows the patient to relax, and this usually involves the examiner supporting the leg with a hand that does not perform clonus. When the limbs move in a passive range of motion, no resistance is felt, but when they move in an active range of motion (e.g. in the upper extremities), this is done by resistance.
Patellar Clonus can be caused by a brain tumour that expels motor neurons in the brain and causes swelling in that area. It can also occur when an injury to a descending motor nerve occurs, which can occur anywhere from the cortex to the spinal cord. Nerves in the brain and spinal cord may also have been damaged, and brain tumors that expel motor neurons and cause the area to swell can lead to cloning. In case of spinal cord injuries, you may have suffered a spinal cord shock that caused a temporary loss of reflexes at the injury stage.
Each spinal cord injury is different, and the severity of the clone can vary greatly from person to person. Some patients may experience the “L’Hermittes” sign, which could be a sign where they describe an electric shock going through their body.
Although the effect of the tremor is normally observed in the cerebellum, it can also be associated with a partial lesion in a descending motor pathway.
Multiple sclerosis affects lower motor neurons, which is why fasciitis is not normally a symptom of the disease. When a limb or muscle is stretched, sensory neurons are sent to the spinal cord. However, if the messages from the brain cannot reach the muscle due to poor blood circulation or a loss of nerve power, it becomes hypersensitive and sends nerve impulses into the muscles on its own. Highly activated alpha neurons increase the sensitivity of the muscle spindles and make them more sensitive to repeated contractions, such as in the spine.
An observational study has shown that SCATS modifies spastic hypertension in patients with SCI-5, suggesting that it is a useful tool for assessing spasmodic activity and spasticity and hypertension in patients with SC I.
Segmental nerve disease with nerve roots T5, T12 and L1: The inhibitory effect of the descending nerve in the gastroznemius muscle in patients with SC I was removed, which led to an increase in muscle spindles and a reduction in spasticity and high blood pressure in these patients.
When it comes to multiple sclerosis, mastering the understanding of the disease means taking care of your Ps and Qs, dosing what is, thwarting the Ts and reciting the ABCs.
Arun Kumar 